Dubai Hotel Industry Essay Example | Topics and Well Written Essays - 6500 words - 1

Dubai Hotel Industry - Essay Example Jumeirah Group tries to stay ahead by offering unique and innovative services through its STAY DIFFERENTTM theme while Rotana’s GET IT RIGHT dictates that every investment must be managed strategically to ensure success before moving to new projectsTatweer has no particular rallying cry but it is responsible for implementing large-scale projects. Industry issues range from an oversupply of hotels, reliance on government support, one-year entertainer visa extension to three-star hotels and terrorist threats. However, all these threats and its effects are mitigated by the initiatives employed by the government which ensures that there will be three times more visitors coming to Dubai by 2010. Analysis indicates that Jumeirah, Rotana and Tatweer will still be largely successful in the future. The degree to which they may differ in success is largely subject to speculation. Nonetheless, all of them will still be enjoying remarkable profits. Dubai, also known as Dubayy, is a city on the northeastern coast of the United Arab Emirates (UAE), and the capital of the emirate of Dubai. The city is divided in half by Dubai Creek, which is actually an inlet of the Persian Gulf. The eastern side of the creek, the traditional city centre, is called Deira (or Dayrah), and the western side is referred to simply as Dubai. Dubai is the chief port and commercial centre of the UAE, and the principal shipping, trading, and communications hub of the Persian Gulf region. Port Rashid, a large artificial port, lies within Dubai on the western side of the creek, and Jebel Ali, the largest artificial port in the world, is located 37 km (23 mi) down the coast to the southwest. Sometimes referred to as â€Å"the Venice of the Gulf,† Dubai is a bustling, cosmopolitan city and a popular tourist destination with a wide selection of upscale shops. In the famous Gold Souk, in Deira, more than 200 shops sell gold jewellery and bullion. Extravagantly designed luxury hotels, some built on artificial islets, line the city’s shore. Dubai hosts an annual high-stakes horse race and major tennis and golf tournaments.     

Cultural struggle Essay Example | Topics and Well Written Essays - 1500 words

Cultural struggle - Essay Example Buy writing Mother Tongue, Amy Tan draws the readers’ attention by giving a reflection of people’s perception that the type and quality of English spoken by an individual represents one’s state of mind. She â€Å"†¦believed that her English reflected the quality of what she had to say. That is because she expressed them imperfectly her thoughts were imperfect† (Tan). Through her statements, readers get to understand that Tan used to look down on her mother due to the quality of English she was using. Further reading Mother Tongue reveals that Amy’s mom is an uneducated woman from China and does not comprehend Standard English. She used to request Amy to make phone calls on her behalf. Amy says that â€Å"she used to have me call people on the phone to pretend I was she †¦ a call to her brokers in New York. I had to get on the phone and say in an adolescent voice that was not very convincing, ‘This is Mrs. Tan.’. The mother as ked Amy to contact the stockbroker and pretend to be her. She, therefore, went ahead to talk to the stockbrokers by paraphrasing her mother’s jumbled sentences into understandable and meaningful statements, to the stockbrokers.Similar captivating information can be derived from the essay Two Kinds, by Amy Tan. In Two Kinds Amy Tan demonstrates that humans carry traumas of their past into their future by dredging them up either intentionally or unconsciously. She uses a Chinese narrator to bring out constant struggles that people go through while trying to find out better understanding and way of existence.

A Literature Review About Mecication Errors Essay Example for Free

A Literature Review About Mecication Errors Essay Introduction An error rate of 5% is acceptable in most industries, however, in the health care industry; one single error can result in death. (Berntsen, 2004, p5) This paper discusses medication errors in relation to pharmacology and drug treatment. It will summarize three academic peer reviewed journal articles, followed by general information in relation to medication errors, the impact of medication errors on client care, strategies to prevent medication errors  and conclude with the relationship to nursing. Summary of Articles Related to Medication Errors. The first article is by Karin Berntsen, 2004, and is entitled How Far Has Health Care Come Since To Err is Human? Exploring Use of Medical Error Data. This is a review of what changes have been made since a medication error report written by the Institute of Medicine was published in 1999. This article depicts how the health care system has changed since this 1999 report was written, and how the information was utilized for our benefit. They concluded that in the USA, medical errors were one of the top 8 leading causes of death. They reported the cost for these errors was between $17 Billion to $29 billion dollars. Until a new report is completed, health care providers will be unaware whether their goals in increasing patient safety were accomplished. The article finalizes that there has been progress in regards to prevention of medication errors and health care leaders feel passionate about increasing patient safety. (Berntsen 2004) The second article is by William N. Kelly, 2004, and is titled Medication Errors: Lessons Learned and Actions Needed and highlights the death of a one year old child who was diagnosed with cancer. She subsequently died, not from the cancer, but from receiving an incorrect dosage of a drug that she was being treated with. This report indicates that medications are systematically checked and balanced and errors are usually caught before a drug is administered to a patient. The article states that problems are not being solved in a timely manner since the industry has been putting band aids on problems that need major surgery.(Kelly 2004). In conclusion, the article questions whether or not they are taking the right approach in preventing errors. Many people are trying to fix this problem however; errors are still made too frequently. (Kelly 2004) The final article is by Rosemary M. Preston, 2004, and is titled Drug Errors and Patient Safety: A Need for Change in Practice. This article presents that errors continue to happen for many reasons. It concentrates upon calculations errors, lack of knowledge of drugs, over/under dosing drugs,  interactions with drugs and food, and legalities regarding drug administration. It also presents recommendations to minimize the risk of drug errors with good communication and honesty. The article closes by stating that nurses should never estimate the skills needed for safe administration of medicines. (Preston 2004) Key aspects: medication errors and their causes. To understand the impact that medication errors have on a patient, we have to understand what a medication error is. According to Health Canada online, a medication error is defined as: Any preventable event that may cause or lead to inappropriate medication use or patient harm while the medication is in the control of the health care professional, patient, or consumer. Such events may be related to professional practice, health care products, procedures, and systems, including prescribing; order communication; product labelling, packaging, and nomenclature; compounding; dispensing; distribution; administration; education; monitoring; and use. [Developed for use by the National Coordinating Council on Medication Error Reporting and Prevention]( http://www.hc-sc.gc.ca/english/index.html) Medication errors occur for a variety of reasons. An error can affect all areas of a health care facility from health care management, staff, physicians, pharmacy and especially patients. Studies have indicated that errors will usually occur when the staff demonstrates signs of fatigue, stress, are over-worked or encounter frequent interruptions and distractions. When physicians display bad handwriting, ineffective communication with patients, and do not educate staff and patients effectively, a medication error is more likely to happen. Poor management can result in more medication errors when there is an emphasis on volume, over service quality. This results in inadequate staffing and disorganization. Medication errors affect all components of the health care environment. (http://www.napra.org/docs/0/95/157-/166.asp) Impact on client care. As disturbing as it sounds, one miniscule error can result in a patients injury or can even lead to their death. According to the American Journal of Medicine, statistics reveal that more than two million American hospitalized patients suffered a serious adverse drug reaction in relation to injury within the 12-month period and, of these, over 100,000 died as a result. http://www4.nationalacademies.org/news) Death and injury is a sad reality to any single error. The government established six rights of drug administration to prevent medication errors and ensure accuracy. These six rights include: Right drug, right dose, Right client, right route, right time and right documentation. (Kozier Erb 2004) Injuries that result from a medication error are called adverse drug events. Usually, these unpleasant effects can be eliminated and injury can be avoided. However, every drug produces harmful side effects, but the severities of these effects vary from individual to individual. These side effects also depend on the drug and the dose given. (Kozier Erb 2004) Health care professionals must report all errors and are accountable for their actions. No matter how insignificant, nurses are taught to document and report all mistakes. When statistics show what types of errors are made, an analysis can be done. This analysis can be used to plan ways to prevent them medication errors. (Berntsen, 2004)When a nurse does not report a mistake, the probability that it will happen again will increase. Medication errors have a huge impact on client care. They can result in death, injury, and result in unwanted effects of drugs. It is our responsibility as nurses to comply with the clients six rights of drug administration, to prevent errors from taking place. Strategies to prevent medication errors. There are many efficient ways to prevent nurses from making an error. To ensure patient safety in all aspects of client care, nurses are taught to think critically, and to problem solve. Nurses use critical thinking to ensure safe, knowledgeable, nursing performance and they must be able to keep up with updated health facts by constantly educating themselves with new information. (Kozier Erb 2004) Critical thinking assists in the prevention of medication errors. The six rights in drug administration help prevent medication errors from occurring. It is important to maintain the highest standards of practice of these rights for a drug to be prepared properly. Failure to adhere to any one of these rights will definitely result in a medication error. (Clayton Stock, 2004) Take your time when preparing medications and research any unknown drugs. Rushing should be avoided when preparing, administering and reading medication labels. Proper research must be done before an unfamiliar drug is administered it to a client. Even when in a rushed emergency situation, drugs should be looked at carefully to know the correct concentration and name of the drug, to prevent injury. (http://www.hc-sc.gc.ca/english/index.html) Labels should be read carefully and accurately. Before a drug is given to a patient, three checks should be done to ensure you are giving the proper drug and dose. In a situation where you are unsure of a drug order, you are expected to refuse the order and clarify it by law. If an individual is unfamiliar with a particular drug, the drug should not be given. (http://www.napra.org/docs/0/95/157-/166.asp) When a label is unclear, do not try not to examine the drug order yourself. Do not ask an associate, or ask for anyone elses interpretation of the drug. To get the correct information, contact the individual who ordered the drug to clarify the label. In order to decrease the chances of error, verify all unclear hand writing, abbreviations, decimal points, decimal places and dosages. (http://www.napra.org/docs/0/95/157-/166.asp) Use of dosage abbreviations should not be used to avoid drug miscalculations. Dosage abbreviations are misinterpreted more often, than any other type of abbreviation. Using standardized abbreviations, would assist in preventing misinterpretation of abbreviations. (Preston 2004) A drug check should be done three times prior to the administration of a drug. The drug label should correspond with the physicians orders. The three checks should be done; Before removing the drug from the shelf or dosage cart, before preparing or measuring the actual prescribed dose, and before replacing the drug on the shelf or before opening a unit dose container, just before administering a dose to a patient. (Clayton Stock, 2004) Do not make assumptions regarding drugs. Physicians, pharmacists, make mistakes and other parts of the health system may be flawed. For example, when documentation shows the patient has no drug allergy, it is wrongful to assume the patient will have no adverse reaction to a new drug. This could result in detrimental results to a clients health. Therefore no assumptions should ever me made. (http://www.ismp.org-/ToolsAllina-Orientation.html) A quiet environment for preparing medications will prevent prescription errors from occurring. Sometimes, nurses are repeatedly interrupted when preparing a medication. Distractions interfere with processing information and decision making. Errors will least likely occur when preparations are done when there are no distractions. (http://www.ismp.org-/ToolsAllina-Orientation.html) When preventing errors, staff must be certain all dosage calculations are correct and clarified. It may be beneficial to ask a colleague to assist you in checking doses, to minimize the chance of miscalculations. Other suggestions to minimize error include; making pre-calculated conversion cards, always use a leading zero before a decimal, never use a zero after the decimal and include indications whenever possible. Miscalculations are preventable if proper methods of inspecting calculations are used. (Preston  2004, p.72) Assess for the effects of drugs to avoid harming a client. A client must be assessed before and after a drug is given. For instance, before giving an oral medication, assess whether the client can swallow or feels nauseated. An appropriate follow up should be done after a medication is administered. It is important to check if the client experienced the desired effect of the drug. Significant abnormal responses to drug should be reported to the physician. (Kozier Erb 2004) Conclusion. To finalize this assignment, medication errors are mistakes that can cause harm to patients and can even result in death. The articles that have been summarized illustrate situations where medication errors have occurred and review what the health care industry is doing to prevent errors. A medication error is preventable and errors can be caused by a variety of reasons. This paper has discussed the impact medication errors have on client care and strategies of how to prevent errors from occurring. As a nurse, this knowledge will assist me in keeping beneficence a priority for client care. References Clayton, Bruce D., BS, RPh, PharmD, and Yvonne N. Stock, MS, BSN, RN. Basic Pharmacology for Nurses. 13th ed. United States of America: Mosby, 2004. Government of Canada Online. (2004, Summer). Retrieved July 18, 2004, from Health Canada Web site: (http://www.hc-sc.gc.ca/english/index.html) Kelly, William N. Medication Errors. Professional Safety 49: 35. Academic Search Elite. EBSCO. Assiniboine Community College. 22 July 2004 . Government of Canada Online. (2004, Summer). Retrieved July 18, 2004, from Health Canada Web site: (http://www.hc-sc.gc.ca/english/index.html) Kozier Erb, Barbara, et al. Fundamentals of Nursing. 7th ed. Upper Saddle River, New Jersey: Pearson Prentice Hall, 2004. Minimizing Medication Errors. (n.d.). In NAPRA: National Association of Pharmacy Regulatory Authorities. Retrieved July 17, 2004, from NAPRA: National Association of Pharmacy Regulatory Authorities Web site: http://www.napra.org/docs/0/95/157/166.asp Preston, Rosemary M. Drug errors and patients safety: the need for a change in practice. British Journal of Nursing (BJN) 13: 72. Academic Search Elite. EBSCO. Assiniboine Community College. 22 July 2004 .

Pantoprazole Compound Discovery and Development

Pantoprazole Compound Discovery and Development Abstract Pantoprazole is an proton pump inhibitor, which inhibits the gastric acid secretion by blocking the proton pump or the H+K+ATPase in the gastric parietal cells of stomach. Pantoprazole was synthesized in the year 1987 and was launched in the year 1994 after development and clinical trials. Development of the lead compound timoprazole and the discovery of H+K+ATPase as an target, were the two most important discoveries which led to the generation of a new class of compounds, the proton pump inhibitors. In this review, the discovery and the stages of development of pantoprazole will be discussed. Introduction The gastric acid is secreted in the stomach by the parietal cells. The gastric parietal cells are known to have three stimulators viz. gastrin, acetycholine and histamine. Acetylcholine and Histamine exert their effect through the M3-Muscarinic and H2-Histamnic receptors respectively. Whereas, gastrin exerts its action via release of histamine. Antagonists of the cholinergic and histaminic receptors were the first agents used for the inhibition of gastric acid secretion. The side effects and low efficacy limited the use of cholinergic receptor antagonists[1] and the histaminic receptor antagonists were the first class of drugs to be clinically used for the treatment of acid related disorders. These classes of drugs were widely developed in the 1970s and 1980s.[1] But, variability in response and phenomenas like acid rebound and tolerance were observed in these class of drugs[2]. Thus there was a need of more effective targets and drugs for the optimal inhibition of gastric acid. This search led to the discovery of a new target, the gastric acid pump- H+K+ATPase and a new class of anti-secretory drugs were born that is the Proton pump inhibitors[2]. Omeprazole was then synthesized in 1979 and was the first clinically used proton pump inhibitor launched in 1988 and then gradually pantoprazole was synthesized in 1986 and launched in 1994. Today pantoprazole is one of the first line drug used in the treatment of acid related disorder. Initiation of research for new compounds: In 1967 at Astra HÃ ¤ssle researcher Ivan -stholm initiated an innovative research project in gastrointestinal field in order to develop anti-secretory agents which could be used in the peptic ulcer diseases[4]. Their first idea was to inhibit the gastric stimulating hormone gastrin[4]. It was known from the various animal experiments that local anesthetic agents of the antrum blocks the release of gastrin. Therefore the researchers at Astra HÃ ¤ssle aimed at synthesizing a local anesthetic compound which could be administered orally and is orally active. But all the available local anesthetic agents were however, protonated in an acidic environment and therefore were inactive, thus the goal was to change the chemical structure of lidocaine which was an established local anesthetic agent of the Astra HÃ ¤ssle itself into a non-basic compound[4]. The shay rat or the gastric fistula rat was used as a screening model. A large number of compounds were synthesized by the researchers, b ut it was found that the anesthetic property of the compound induced toxic effects. The chemical development finally ended with compounds including carbamates which were devoid of local anesthetic properties. Carbamates were found to be very effective inhibitors of gastric acid secretion in rat models but were rather ineffective in dog.[6-7] The most effective carbamate compound was H81/75 but however in 1971-72, when it was tested in humans it was found to be completely ineffective. Instead of reviving this local anesthetic lead, the researchers undertook a literature search to look for new approaches. In 1972, the researchers found an abstract from an Hungarian pharmacological meeting in which a new anti-secretory agent called CMN-131 was described.CMN-131 was synthesized by French company Servier.[4,5] In this abstract it was reported that CMN-131 induced inhibition of stimulated gastric acid secretion in rats as well as anesthetized dogs. But due to severe toxicological problems the research on this drug never continued.[4] By this time in 1973, Smith-Kline and French announced the development of Cimetidine, worlds first H2 receptor antagonist which inhibited the gastric acid secretion by blocking histaminic receptors[1]. Based on the structure of cimetidine, a benzimidazole ring was added to the structure of CMN-131and was tested on animal models this new compound was named as H124/26 .[4]This sulphide compound was then modified for stabilization into its su lfoxide analogue and thus a new compound called as Timoprazole was born which was found to be a potent inhibitor of gastric acid secretion. But, however timoprazole was found to show toxicities in the thyroid gland. It causes enlargement of thyroid gland, the possible reason for this toxicity was that the timoprazole inhibits the iodine uptake. Thus timoprazole was not further developed and it served as a lead compound for the development of new anti-secretory agents. Uptill now the target of timoprazole was unknown. Discovery of H+K+ ATPase: In 1977, George Sachs and John Forte discovered H+K+ ATPase pump commonly known as the proton pump or the gastric acid pump[3.4]. From the experiments carried out on hog gastric mucosa, they showed that the exchange of H+ and K+ were responsible for the regulation of the gastric acid secretion and they also suggested that this was the terminal step in the acid secretory process of the parietal cell wall[4]. When acid secretory membranes are isolated from the parietal cells, they round up and form closed vesicles containing H+K+ ATPase. On the basis of imunohistological data from various organs with the help of antibodies against a crude preparation from the secretory membranes of the parietal cells, Sachs showed that the proto pump was localized in the gastric parietal cells.[4] This immunohistological data not only revealed strong immunoreactivity in the parietal cell region of the stomach, but also revealed some acitivity in the thyroid gland.[5] Target identification: On the basis of various pharmacological methods, like the isolated guinea pig atrium, it was found that timoprazole was neither an H2-histaminic receptor antagonist nor an anti-cholinergic drug. Furthermore there were no evidences supporting any anti-gastrin activity of the compound.[4,5] Therefore, though timoprazole inhibited the gastric acid secretion in various animal based models but its exact mechanism and site of action due to which it can account for its anti-secretory activity was yet to be identified. During this time, the proton pump was discovered and there were evidences that the activation of this newly discovered proton pump, present in the secretory membranes of the stomach parietal cells, was the final step of the gastric acid secretion. Also, the imunohistological data obtained using antibodies reveled strong immunoreactivity in the parietal cell region of the stomach and also some activity in the thyroid gland. On the basis of these facts coupled with the knowledge of the side effects of timoprazole on the thyroid gland, discussed earlier, raised an intriguing question in the minds of the scientists that could H+K+ ATPase, be the target of site of action of timoprazole. Research was initiated in this area in parallel to the further development of the benzimidazole compounds. With the help of the various pharmacological techniques such as the isolated gastric vesicles, it was indeed shown that the substituted benzimidazoles inhibited the gastric acid secretion by the inhib ition of the H+K+ ATPase pump. Studies showed that the pre-incubation of isolated vesicles with substituted imidazoles resulted in inhibition of gastric acid secretion only when the conditions were acidic[4,5]. This was really a breakthrough finding. This finding was further verified in experiments where the compound solvent was acidified[4]. All these facts and findings were the first indication that the substituted benzimidazoles had to be probably be transformed in other forms in order to bring about the active inhibition of the proton pump. Protonation of the compound was the first step in the transformation of compounds. These findings were followed by a series of experiments using various different types of test systems, in order to study the interactions of substituted benzimidazoles with the H+K+ ATPase pump. Several binding studies were carried out with substituted benzimidazoles which showed specific binding to the H+K+ ATPase.[3,4] All the studies and findings showed that the substituted benzimidazoles inhibited the gastric acid secretion by binding to the H+K+ ATPase pump and thus inhibiting its action. Optimization of timoprazole: Due to the various toxicological effects of timoprazole on the thyroid gland due to the inhibition of the iodine uptake, timoprazole was not suitable for further development. Therefore the researchers were in search of a new compound and a new possible approach. In order to optimize the lead compound timoprazole various studies were carried out, so that an compound devoid of toxicities could be developed. A literature search of the chemistry of thiourea compounds showed few substituted mercapto-benzimidazoles having no effect on the iodine uptake by the thyroid[4,5]. Thse substituted mercapto-benzimidazoles analogues were introduced into the structure of timoprazole. Various tests and experiments showed that the above analogue of timoprazole had a considerable anti-secretory actitvity and also was devoid of any inhibitory action on the uptake of iodine. This potent anti-secretory compound obtained after the introduction of mercapto-benzimidazole substituents in the structure of timoprazole was named as picoprazole.[5,4] The first toxicological studies of picoprazole showed necrotizing vasculitis in the small intestine of dogs[4]. However it was later found out that the toxic effect of picoprazole was an non-drug related phenomenon. The second toxicological studies carried out with picoprazole was rather successful. Picoprazole was then tested in human volunteers, where it showed very potent anti-secretory activity with a long duration of action.[4,5] Development of pantoprazole: As discussed earlier, these compounds were only effective in inhibition of gastric acid secretion, if an only if the ATPase was making acid. As this compound was a weak base the steps that were thought then to result in inhibition of ATPase activity and acid secretion involved accumulation of the compound in the acid space of the isolated or intact gastric vesicles or in the parietal cell canalicullis during H+ transport, followed by a conversion of compound to its active form to account for acid dependence.[1] The conversion of compound to its active form is acid dependent. It was then postulated that these compounds acted as pro-drugs which can only react with the H+/K+ ATPase,if they are converted into their active form in an acid dependent manner. The active form of this compounds are the sulfenic acid or sulfenamide form. Further studies showed that the final structure of the compound generated in the acidic solution was a result of tetracyclic planar rearrangement of the compou nd, which leads to compounds containing a highly -SH reactive sulfenamide group.[1] However it is not clear whether the sulfenamide or the sulfenic acid or its dehydro form is responsible for binding to the H+/K+ ATPase covalently. In order optimize the acid stability of the lead compound and to generate selectivity for maximal accumulation at the site of action and for proper activation in the acidic space of the parietal cells, chemists changed and introduced new substituents on the heterocyclic ring of the lead compound which lead to the development and synthesis of Omeprazole in the year 1979[5]. It was found to be the most powerful inhibitor of stimulated gastric acid release[5]. Omeprazole was devoid of toxicities. Omeprazole was launched in 1988 and it was the first clinically used proton pump inhibitor. In order to develop more acid stable and effective compounds various modifications were done and in 1986 Byk Guilden synthesized Pantoprazole.[3] It was tested in both in vivo and in vitro and was found to be a potent anti secretory agent. studies on human volunteers was successful and it also suggested that pantoprazole had greater acid stability and target selectivity than omeprazole[2]. In addition its pharmacokinetic and metabolic profile was also different[2]. In 1987 sodium salt of pantoprazole was synthesized as the salt was more stable, more soluble and was more compatible with other excipients used in the formulation finally after seven years of clinical development pantoprazole was launched in 1994 for the first time in Germany.

Karl Marx Essay -- essays research papers

Karl Marx was born on May 5, 1818, in Trier, Prussia. He attended the university of Bonn and later the university at Berlin, where he studied in law, while majoring in history and philosophy. After his education, Marx associated himself with the "Left Hegelians," along with Bruno Bauer, which were a group who formed atheistic and revolutionary ideas from Hegel's philosophy. The Young Hegelians practiced philosophical idealism. Here he first meet Arnold Ruge and Ludwig Feuerbach. In 1842 Marx and Bruno Bauer were asked to contribute to the Rheinische Zeitung, a German paper, in Cologne. At the time Marx started, the paper had only 400 subscribers. Marx in October of 1842, became editor-in-chief, and decided to move from Bonn to Cologne. As the paper became more and more revolutionary and widely read, the government decided to censor, and eventually suppress it. The paper was banned in March of 1843. At this time, it had more than 3,400 subscribers from all over Germany. Karl Marx was married to his childhood friend Jenny von Westphalen, in 1843. Later in the fall of that year Marx along with another Left Hegelian, Arnold Ruge, moved to Paris and began publication of a radical journal entitled Deutsch-Franzosische Jahrbucher. Due to the problems in publishing such a radical paper, only one issue appeared. Karl met his closest friend in September of 1844, when Frederick Engels arrived in Paris. Together they participated in the activities of many revolutionary communities. They formed the theory and ideas of revolutionary proletarian socialism, also known as communism. Also in 1844, Marx wrote a revolutionary book called the Holy Family. It is a materialist view of the history of man. Basically, it was a critique on his former philosophy group, "The Young Hegelians." It expressed the view of history being mans activities. "‘History' is not, as it were, a person apart, using man as a means to achieve its own aims; history is nothing but the activity of man pursuing his own aims." Finally in 1845 Marx was banished from Paris as a dangerous revolutionary. He wrote satirical poems for revolutionary-democrats. The paper, "Vorwà ¤rts", was attack by reactionary papers asking for government banning or censorship, but instead they banned Marx from Paris. He decided to head for Brussels, where he and Engels joined, in 1847,... ...pital is placed on exploitation of wage labor. Marx discovered the fact that it is not labor itself, but labor power that is the commodity which the worker sells and the capitalist purchases. This is the key discovery for the connection for the determination of the commodity value by labor. Capital also establishes two main points. The first is that the capitalist can increase the rate of surplus value by prolonging the working day. This is absolute surplus value. The second is by increasing the intensity of the working day. This is relative surplus value. Marx covers every aspect of capitalist economy, and depicts every flaw. Though never completed, it is the work of his life time. On December 2, 1881, His lifelong wife passed away. Marx's health declined due to his strenuous work with the International and the Capital. And just more than a year later Karl Marx died peacefully at his home. Both were buried at Highgate Cemetery in London, where they still lie today. Marx has affected nearly all aspects, whether it be economy, politics, natural science, Darwinism, or even philosophy. His impacts have positively changed the world even still today, Marx's ingenious all around us.

ATVs: A Mans World :: essays research papers fc

ATV’s: A Man’s World   Ã‚  Ã‚  Ã‚  Ã‚  Propaganda is everywhere. It is on the television, in the newspapers, magazines, and the internet. Everywhere you turn, it’s there. The problem with this is that people merely dismiss it as truth. When people see propaganda, they don’t â€Å"see† propaganda; they see a nice new sport-utility vehicle with a â€Å"powerful engine† and â€Å"cool rims†. When an advertisement uses propaganda, it’s over-looked and the reader is caught in the trap laid out for him. People see everything the designer of that particular advertisement wants them to see. The advertisements selected and interpreted for this assignment are directed towards male’s ages 18 to 27 in the United States. They use three different forms of propaganda to send the same message; â€Å"Buy this, it’s fun and your friends will be so jealous.†   Ã‚  Ã‚  Ã‚  Ã‚  The first advertisement, for the Arctic Cat 650, uses the glittering generalities technique to lure people into buying their product. It depicts a young man riding their product, a massive ATV, with a helmet and ATV eyewear. He is covered in mud from head to toe and seems to carry this â€Å"I’m better than you are.† persona. It seams to me that simply because he is riding this particular product, he feels as though he is better than any who is not riding the same ATV. The glittering generalities technique uses specific words to make us believe that their product is better for us or will make us happier than their competitors’ similar product. This advertisement uses many â€Å"glittering† words and phrases to make their product more appealing to those targeted. Some of these phrases include ‘new and improved for 2005’ and ‘the amazing’. These glittering generalizations could be said about any all-terrain vehic le. The problem with generalizations is that there is no hard evidence to back it up, they simply state what the designer wants them to state. However, the normal reader of this advertisement would never question the truth of the words stated therein. Other generalizations in this advertisement include, ‘the most powerful ATV on the market’. How can they claim this without giving proof of their claim? Simple, they are using propaganda to stretch the truth.   Ã‚  Ã‚  Ã‚  Ã‚  Another advertisement uses testimonials or endorsements to sell their product, the Kawasaki Brute Force. This ad shows their product ahead of the pack in an actual race. He is just crossing the finish line as the picture was taken.

A Moment That Changed My Life Essay

This is my creative essay for the Film Program at NYU. The prompt was: â€Å"Introduce yourself. Describe an unforgettable event in your life and how it changed your perception of yourself or the view of someone close to you. This event can be dramatic and/or comedic. The assignment may be written as a short story in the first person or as an essay. † My one fear is that my essay is hard to understand. So any feedback on how it reads to other people would be a huge help. And also any gramatical errors or anything like that that you find would be a help as well. I’m probably mailing this out today so please, any advice would be much appriciated. Thanks. How did I get here? How did this happen. I sat staring through the open car door as it dangled there in front of my eyes. How had I not seen it before; how had it never caught my eye? It hit me so fast, like a wave that unexpectedly overtakes you as you walk along the beach. â€Å"It’s really happening,† I thought to myself. I am no longer a child. I am terrified. And it’s not fear that troubles me, it’s realization. It’s the realization that my life will simply just keeps moving along and I have no say in the matter. It’s the realization that I no longer have what seems like all the time in the world to figure out what I want to do with that life; what I want to be, what I want to leave behind after I’m gone. It’s finally time for me to stop thinking but rather go out and do the things I’ve dreamed of; the things I’ve only wondered at, baffled at, as I lay alone in my bed at night. It had never felt real until now. I stared at it as it hung there, as it hung unaware of the meaning it gave to me at this moment. Those few seconds stretched for hours, stretched to encompass the span of a lifetime in the flash of a blinking eye. And that’s what it all now felt like; a blink, a moment, a flash. Is that what it will feel like at the end? A flash? When I was a little girl, my father took me to the fair. He bought me a wristband so that I could go on all of the rides without having to fumble with tickets. He told me, â€Å"Darling, you can do anything you want here, just say the word. † To a young child, hearing those words was like being given the keys to the world; being given complete freedom. To me, freedom was a precious thing. I craved it; desired it above all else. But you see, in my eyes, freedom was simply having whatever you want, when you want, and not having to do anything you don’t want to get it. In my eyes, freedom was a perfect world, but only for me. Of course over time I was persuaded from these views by opening my eyes to the hard work and responsibility involved in having â€Å"freedom. † As I grew older I earned more privileges. I worked to make my own money, began driving, found myself a wonderful boy, and even began preparing for college; dreaming of going out on my own and making a name for myself in this wide world. And for a while the illusion was indeed convincing. I allowed myself to believe that over time I would gain more control in my life and that that control would one day become absolute. But it was still just an illusion, a dream; an idea of a perfect world, my perfect world. Eventually I would realize that the one thing which I thought could bring me my absolute freedom is the one thing that will forever hold it back. Time. Time is the one thing which binds everyone. Even the most wealthy, most powerful people in the world will die someday. Do you think they chose that for themselves? Do you think they wouldn’t change it if they could? You could have the whole world in the palm of your hand, but the reality remains; everyone is born, everyone grows older, and everyone will eventually die. Our time is limited. That can’t be changed, just accepted. So staring, I sat as it hung limply outside the open car door. How had I never seen it before? How had it never caught my eye? How had it all happened so fast? The lines, those small cracks, they hung loosely together over the weakened bone below. I sat staring, staring at my father’s aged hand that hung at his side as he stood outside the car conversing with someone who, at the moment, held no importance to me. Never before had I noticed those lines, those cracks, those wrinkles which spread out like ripples on the surface of a pond. Never before had I noticed how thin the skin appeared, how stretched, or even how loosely the veins spread underneath. For the first time I was looking at my father; a man in his fifties, hardened by years of ceaseless working. For the first time in a long time, I was seeing what was actually there and not just what my eyes were telling me to see based off of what they had previously known. For the first time, I realized I was growing up. For the first time, I felt the hold that time has on me. For the first time, I thought â€Å"I am almost eighteen years old. What have I done with that time? † What had I done? I hated the thought. I dreaded the question. That question to which my answer would speak of nothing of importance. At this time, what troubled me most was not the fact that I – and everyone around me – were getting older. At this time, what troubled me most was the fact that- in all that time of growth- the things which I had done served no greater purpose than to merely entertain; to entertain myself or the requests of others, teachers, my parents. I thought, â€Å"Has all this time been wasted? † Time goes by so fast; faster than even I can sometimes realize. The funny thing is, the older I get, as the years I have left steadily decrease, I can’t help feeling time’s steady increase. And this only leads me to further wonder; if these eighteen years have gone by so rapidly, as if they were nothing, will it not feel as though tomorrow I will find myself an old woman? Is it so unlikely? Is it so unlikely that, if the lifestyle I have I keep, I will wake up on that day and again have to question if the time I had been given had been squandered away in futile activity? A sad day it would be to lay in remembrance of your life and realize that the days you have spent held no purpose or meaning beyond to merely serve yourself and your own selfish comforts. A sad day it would be to realize that you have lived your life well and comfortably, yes, but never with purpose. And it was then, for a moment, that I was torn from my thoughts as my father at last settled back into the car and began to drive us home. I looked over at the man who had given me everything; my life, my childhood, and now this much needed revelation. Unbeknown to him, in an instant, he had changed my views of this world and of the life I was leading. In an instant, he had given me what was needed for me to stop waiting around, stop simply thinking of how my life will be, and start living, actually living. In that moment, in that rough weathered hand, I saw my future. I saw the effects that time would one day have on me. I saw the old age that would one day overtake me. I saw myself upon that final hour, laying, awaiting that final breath. But what I did not see was how I would greet that end. Would it be with peaceful acceptance or mournful detest? The real question is; when that final hour comes, when I take that final breathe, will the death that takes me be taking a soul that has truly lived? Will I have truly lived? I sit, looking at my father’s hand as he steers us home. I sit, not hoping for, but planning the things I am going to do, the things which I will do, today and tomorrow and all the days after. The things which will, in purpose, serve greater causes than to just simply allow me to go on drifting along in my life. For from now on I will do nothing but dream and create. I will do nothing but share with others my thoughts, my words, my ideas. I will do nothing but strive with all I have to make a difference in, if even in the smallest way, the life of another, the ways of a people, the turning of the world. From now on I will nothing but truly just live.